p53 at the Intersection of Cardiac Fibroblast Proliferation and Activation: Answers and Questions-Reference-Cited by-同舟云学术

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p53 at the Intersection of Cardiac Fibroblast Proliferation and Activation: Answers and Questions

Published:2023-07-21 Issue:3 Volume:133 Page:288-290
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circulation Research

Author:

Ghigo Alessandra¹^ORCID,Ameri Pietro²³^ORCID

Affiliation:

1. Department of Molecular Biotechnology and Health Sciences, Molecular Biotechnology Center “Guido Tarone,” University of Torino, Italy (A.G.).

2. Department of Internal Medicine, University of Genova, Italy (P.A.).

3. Cardiovascular Disease Unit, Cardiac, Thoracic and Vascular Department, IRCCS Ospedale Policlinico San Martino – IRCCS Italian Cardiology Network, Genova, Italy (P.A.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference13 articles.

1. Cardiac fibrosis

2. Therapeutic targets for cardiac fibrosis: from old school to next-gen

3. Single-cell expression profiling reveals dynamic flux of cardiac stromal, vascular and immune cells in health and injury

4. Specialized fibroblast differentiated states underlie scar formation in the infarcted mouse heart

5. Resident fibroblast expansion during cardiac growth and remodeling

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Indoxyl sulphate‐initiated activation of cardiac fibroblasts is modulated by aryl hydrocarbon receptor and nuclear factor‐erythroid‐2‐related factor 2;Journal of Cellular and Molecular Medicine;2024-03-20

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