Ca 2+ Signaling Domains Responsible For Cardiac Hypertrophy and Arrhythmias
Author:
Affiliation:
1. From the Temple University School of Medicine, Philadelphia, Pa.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Physiology
Link
https://www.ahajournals.org/doi/pdf/10.1161/CIRCRESAHA.109.193821
Reference16 articles.
1. Calcium Cycling and Signaling in Cardiac Myocytes
2. Does Contractile Ca 2+ Control Calcineurin-NFAT Signaling and Pathological Hypertrophy in Cardiac Myocytes?
3. Multiple downstream proarrhythmic targets for calmodulin kinase II: Moving beyond an ion channel-centric focus
4. miR-1 Overexpression Enhances Ca 2+ Release and Promotes Cardiac Arrhythmogenesis by Targeting PP2A Regulatory Subunit B56α and Causing CaMKII-Dependent Hyperphosphorylation of RyR2
5. The Ca V 3.2 T-Type Ca 2+ Channel Is Required for Pressure Overload–Induced Cardiac Hypertrophy in Mice
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