Developmental Heterogeneity of Cardiac Fibroblasts Does Not Predict Pathological Proliferation and Activation-Reference-Cited by-同舟云学术

Developmental Heterogeneity of Cardiac Fibroblasts Does Not Predict Pathological Proliferation and Activation

Published:2014-09-12 Issue:7 Volume:115 Page:625-635
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circ Res

Author:

Ali Shah R.¹,Ranjbarvaziri Sara¹,Talkhabi Mahmood¹,Zhao Peng¹,Subat Ali¹,Hojjat Armin¹,Kamran Paniz¹,Müller Antonia M.S.¹,Volz Katharina S.¹,Tang Zhaoyi¹,Red-Horse Kristy¹,Ardehali Reza¹

Affiliation:

1. From the Departments of Pathology and Developmental Biology, Institute for Stem Cell Biology and Regenerative Medicine, Stanford University School of Medicine, CA (S.R.A.); Department of Internal Medicine, Division of Cardiology, and Broad Stem Cell Research Center, University of California Los Angeles School of Medicine (S.R., M.T., P.Z., A.S., A.H., P.K., Z.T., R.A.); and Division of Blood and Marrow Transplantation, Department of Medicine (A.M. S.M.) and Department of Biology (K.S.V., K.R.-H.),...

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference36 articles.

1. The origin of fibroblasts and mechanism of cardiac fibrosis

2. Fibroblast-specific protein 1 identifies an inflammatory subpopulation of macrophages in the liver

3. Hematopoietic origin of fibroblasts/myofibroblasts: its pathophysiologic implications

4. Endothelial-to-mesenchymal transition contributes to cardiac fibrosis

5. Bone marrow-derived myofibroblasts contribute functionally to scar formation after myocardial infarction

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