Triple Twist Theory of Rho Inhibition by the Angiotensin II Type 2 Receptor
Author:
Affiliation:
1. From the Cardiovascular Research Center and Department of Physiology, Temple University School of Medicine, Philadelphia, Pa.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Physiology
Link
https://www.ahajournals.org/doi/pdf/10.1161/CIRCRESAHA.108.177295
Reference28 articles.
1. Dual Effects of Angiotensin II Type 2 Receptor on Cardiovascular Hypertrophy
2. The Angiotensin II Type 2 Receptor Causes Constitutive Growth of Cardiomyocytes and Does Not Antagonize Angiotensin II Type 1 Receptor–Mediated Hypertrophy
3. Cardiovascular and Renal Regulation by the Angiotensin Type 2 Receptor
4. Angiotensin II cell signaling: physiological and pathological effects in the cardiovascular system
5. Emerging Concepts of Regulation of Angiotensin II Receptors
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1. Valsartan Upregulates Kir2.1 in Rats Suffering from Myocardial Infarction via Casein Kinase 2;Cardiovascular Drugs and Therapy;2015-06
2. Suppression of Ventricular Arrhythmias After Myocardial Infarction by AT1 Receptor Blockade: Role of the AT2 Receptor and Casein Kinase 2/Kir2.1 Pathway;Cardiovascular Drugs and Therapy;2015-06
3. Angiotensin II type-1 receptor regulates RhoA and Rho-kinase/ROCK activation via multiple mechanisms. Focus on “Angiotensin II induces RhoA activation through SHP2-dependent dephosphorylation of the RhoGAP p190A in vascular smooth muscle cells”;American Journal of Physiology-Cell Physiology;2009-11
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