GRK5-Mediated Exacerbation of Pathological Cardiac Hypertrophy Involves Facilitation of Nuclear NFAT Activity-Reference-Cited by-同舟云学术

GRK5-Mediated Exacerbation of Pathological Cardiac Hypertrophy Involves Facilitation of Nuclear NFAT Activity

Published:2014-12-05 Issue:12 Volume:115 Page:976-985
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circ Res

Author:

Hullmann Jonathan E.¹,Grisanti Laurel A.¹,Makarewich Catherine A.¹,Gao Erhe¹,Gold Jessica I.¹,Chuprun J. Kurt¹,Tilley Douglas G.¹,Houser Steven R.¹,Koch Walter J.¹

Affiliation:

1. From the Center for Translational Medicine, Thomas Jefferson University, Philadelphia, PA (J.E.H., J.I.G.); and Center for Translational Medicine (J.E.H., L.A.G., E.G. J.I.G., J.K.C., D.G.T., W.J.K.) and Cardiovascular Research Center (C.A.M., S.R.H.), Temple University School of Medicine, Philadelphia, PA.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

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4. Characterization Of Lymphocyte beta2-Adrenoceptor Signalling In Patients With Left Ventricular Volume Overload Disease

5. Differential functional expression of human myocardial G protein receptor kinases in left ventricular cardiac diseases

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