Atrogin1/MAFbx
Author:
Affiliation:
1. From the Department of Cell Biology, Harvard Medical School, Boston, MA.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Physiology
Link
https://www.ahajournals.org/doi/pdf/10.1161/CIRCRESAHA.111.248872
Reference19 articles.
1. Endogenous Muscle Atrophy F-Box Mediates Pressure Overload–Induced Cardiac Hypertrophy Through Regulation of Nuclear Factor-κB
2. Proteasome inhibition promotes regression of left ventricular hypertrophy
3. Atrogin-1, a muscle-specific F-box protein highly expressed during muscle atrophy
4. Multiple types of skeletal muscle atrophy involve a common program of changes in gene expression
5. Rapid disuse and denervation atrophy involve transcriptional changes similar to those of muscle wasting during systemic diseases
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