Severe Hyperhomocysteinemia Promotes Bone Marrow–Derived and Resident Inflammatory Monocyte Differentiation and Atherosclerosis in LDLr/CBS-Deficient Mice-Reference-Cited by-同舟云学术

Severe Hyperhomocysteinemia Promotes Bone Marrow–Derived and Resident Inflammatory Monocyte Differentiation and Atherosclerosis in LDLr/CBS-Deficient Mice

Published:2012-06-22 Issue:1 Volume:111 Page:37-49
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circ Res

Author:

Zhang Daqing¹,Fang Pu¹,Jiang Xiaohua¹,Nelson Jun¹,Moore Jodene K.¹,Kruger Warren D.¹,Berretta Remus M.¹,Houser Steven R.¹,Yang Xiaofeng¹,Wang Hong¹

Affiliation:

1. From the Department of Pharmacology (D.Z., P.F., X.J., J.N., X.Y., H.W.), Cardiovascular Research Center (P.F., X.J., J.N., R.M.B., S.R.H., X.Y., H.W.), and Thrombosis Research Center (H. Wang), Temple University School of Medicine (J.K.M.), Philadelphia, PA; Department of Cardiology, Shengjing Hospital of China Medical University, Shenyang, People's Republic of China (D.Z.); Fox Chase Cancer Center, Philadelphia, PA (W.D.K.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference40 articles.

1. Homocysteine inhibits endothelial cell growth via DNA hypomethylation of the cyclin Agene

2. Hyperhomocystinemia Impairs Endothelial Function and eNOS Activity via PKC Activation

3. Hyperhomocysteinemia accelerates atherosclerosis in cystathionine β-synthase and apolipoprotein E double knock-out mice with and without dietary perturbation

4. Hyperhomocysteinemia inhibits post-injury reendothelialization in mice

5. Hyperhomocysteinemia Decreases Circulating High-Density Lipoprotein by Inhibiting Apolipoprotein A-I Protein Synthesis and Enhancing HDL Cholesterol Clearance

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