Antigen-Dependent and Antigen-Independent Pathways Modulate CD4 + CD28 null T-Cells During Atherosclerosis-Reference-Cited by-同舟云学术

Antigen-Dependent and Antigen-Independent Pathways Modulate CD4 + CD28 null T-Cells During Atherosclerosis

Published:2012-07-06 Issue:2 Volume:111 Page:
ISSN:0009-7330
Container-title:Circulation Research
language:en
Short-container-title:Circ Res

Author:

Ammirati Enrico¹,Monaco Claudia²,Norata Giuseppe Danilo³

Affiliation:

1. San Raffaele Scientific Institute and Vita-Salute University Milan, Italy Heart Transplantation Division Ospedale Niguarda Ca' Granda Milan, Italy (Ammirati)

2. Kennedy Institute of Rheumatology Division Imperial College School of Medicine London, UK (Monaco)

3. Department of Pharmacological Sciences Università degli Studi di Milano Milan, Italy Center for the Study of Atherosclerosis Bassini Hospital Cinisello Balsamo, Italy The Blizard Institute Centre for Diabetes Barts and The London School of Medicine & Dentistry Queen's Mary University London, UK (Norata)

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology

Reference7 articles.

1. High Levels of Costimulatory Receptors OX40 and 4-1BB Characterize CD4 + CD28 null T Cells in Patients With Acute Coronary Syndrome

2. Expansion of T-Cell Receptor ζ dim Effector T Cells in Acute Coronary Syndromes

3. TCR ζ-chain downregulation: curtailing an excessive inflammatory immune response

4. TCRζdimlymphocytes define populations of circulating effector cells that migrate to inflamed tissues

5. Effector Memory T cells Are Associated With Atherosclerosis in Humans and Animal Models

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