Transient Outward K + Current (I to ) Underlies the Right Ventricular Initiation of Polymorphic Ventricular Tachycardia in a Transgenic Rabbit Model of Long-QT Syndrome Type 1-Reference-Cited by-同舟云学术

Transient Outward K + Current (I to ) Underlies the Right Ventricular Initiation of Polymorphic Ventricular Tachycardia in a Transgenic Rabbit Model of Long-QT Syndrome Type 1

Published:2018-06 Issue:6 Volume:11 Page:
ISSN:1941-3149
Container-title:Circulation: Arrhythmia and Electrophysiology
language:en
Short-container-title:Circ Arrhythm Electrophysiol

Author:

Choi Bum-Rak¹,Li Weiyan¹,Terentyev Dmitry¹,Kabakov Anatoli Y.¹,Zhong Mingwang²,Rees Colin M.²,Terentyeva Radmila¹,Kim Tae Yun¹,Qu Zhilin³,Peng Xuwen⁴,Karma Alain²,Koren Gideon¹

Affiliation:

1. Cardiovascular Research Center, Division of Cardiology, Rhode Island Hospital, Warren Alpert Medical School of Brown University, Providence (B.-R.C., W.L., D.T., A.Y.K., R.T., T.Y.K., G.K.).

2. Department of Physics, Northeastern University, Boston MA (M.Z., C.M.R., A.K.).

3. Department of Medicine (Cardiology), University of California, Los Angeles (Z.Q.).

4. Department of Comparative Medicine, Pennsylvania State University College of Medicine, Hershey (X.P.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference37 articles.

1. Positional cloning of a novel potassium channel gene: KVLQT1 mutations cause cardiac arrhythmias

2. Comparison of clinical and genetic variables of cardiac events associated with loud noise versus swimming among subjects with the long QT syndrome

3. Cellular basis for long QT, transmural dispersion of repolarization, and torsade de pointes in the long QT syndrome

4. Probing the Contribution of I Ks to Canine Ventricular Repolarization

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