Unfolded Protein Response Regulates Cardiac Sodium Current in Systolic Human Heart Failure-Reference-Cited by-同舟云学术

Unfolded Protein Response Regulates Cardiac Sodium Current in Systolic Human Heart Failure

Published:2013-10 Issue:5 Volume:6 Page:1018-1024
ISSN:1941-3149
Container-title:Circulation: Arrhythmia and Electrophysiology
language:en
Short-container-title:Circ Arrhythm Electrophysiol

Author:

Gao Ge¹,Xie An¹,Zhang Jianhua¹,Herman Amanda M.¹,Jeong Euy-Myoung¹,Gu Lianzhi¹,Liu Man¹,Yang Kai-Chien¹,Kamp Timothy J.¹,Dudley Samuel C.¹

Affiliation:

1. From the Lifespan Cardiovascular Research Center, the Warren Alpert School of Medicine, Brown University and Providence Veterans Administration Medical Center, Providence, RI (G.G., A.X., E.-M.J., L.G., M.L., K.-C.Y., S.C.D.); and Division of Cardiovascular Medicine, Department of Medicine, University of Wisconsin School of Medicine and Public Health, Madison, WI (J.Z., A.M.H., T.J.K.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference33 articles.

1. Increased late sodium current in myocytes from a canine heart failure model and from failing human heart

2. A novelSCN5Amutation associated with idiopathic ventricular fibrillation without typical ECG findings of Brugada syndrome

3. The Brugada syndrome

4. High Risk for Bradyarrhythmic Complications in Patients With Brugada Syndrome Caused by SCN5AGene Mutations

5. Primary structure and functional expression of the human cardiac tetrodotoxin-insensitive voltage-dependent sodium channel.

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