Atherosclerosis Modified
Author:
Affiliation:
1. From the Departments of Biochemistry and Comparative Biosciences, University of Wisconsin-Madison, Madison, Wis.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Physiology
Reference19 articles.
1. Phenotypes of Patients with “Simple” Mendelian Disorders Are Complex Traits: Thresholds, Modifiers, and Systems Dynamics
2. Disruption of the 12/15-lipoxygenase gene diminishes atherosclerosis in apo E–deficient mice
3. Lack of macrophage fatty-acid–binding protein aP2 protects mice deficient in apolipoprotein E against atherosclerosis
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