MnSOD Deficiency Increases Endothelial Dysfunction in ApoE-Deficient Mice-Reference-Cited by-同舟云学术

MnSOD Deficiency Increases Endothelial Dysfunction in ApoE-Deficient Mice

Published:2006-10 Issue:10 Volume:26 Page:2331-2336
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Ohashi Masuo¹,Runge Marschall S.¹,Faraci Frank M.¹,Heistad Donald D.¹

Affiliation:

1. From the Cardiovascular Center and Department of Internal Medicine and Pharmacology, Roy J. and Lucille A. Carver College of Medicine (M.O., F.M.F., D.D.H.) and VA Medical Center (D.D.H.), The University of Iowa, Iowa City; and the Department of Internal Medicine, University of North Carolina at Chapel Hill (M.S.R.).

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference35 articles.

1. Mitochondrial disease in mouse results in increased oxidative stress

2. Mitochondrial Genetics: A Paradigm for Aging and Degenerative Diseases?

3. Dilated cardiomyopathy and neonatal lethality in mutant mice lacking manganese superoxide dismutase

4. Increased mitochondrial oxidative stress in the Sod2 (+/-) mouse results in the age-related decline of mitochondrial function culminating in increased apoptosis

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