Expression of Fractalkine (CX3CL1) and its Receptor, CX3CR1, Is Elevated in Coronary Artery Disease and Is Reduced During Statin Therapy-Reference-Cited by-同舟云学术

Expression of Fractalkine (CX3CL1) and its Receptor, CX3CR1, Is Elevated in Coronary Artery Disease and Is Reduced During Statin Therapy

Published:2005-12 Issue:12 Volume:25 Page:2567-2572
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Damås Jan K.¹,Boullier Agnes¹,Wæhre Torgun¹,Smith Camilla¹,Sandberg Wiggo J.¹,Green Simone¹,Aukrust Pål¹,Quehenberger Oswald¹

Affiliation:

1. From the Department of Medicine (J.K.D., A.B., S.G., O.Q.), University of California, San Diego, La Jolla; and the Research Institute for Internal Medicine (J.K.D., T.W., C.S., W.J.S., P.A.), the Department of Cardiology (T.W.), and the Section of Clinical Immunology and Infectious Diseases (P.A.), Rikshospitalet, University of Oslo, Norway.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference32 articles.

1. Atherosclerosis

2. Innate and Adaptive Immunity in the Pathogenesis of Atherosclerosis

3. Decreased lesion formation in CCR2−/− mice reveals a role for chemokines in the initiation of atherosclerosis

4. A leukocyte homologue of the IL-8 receptor CXCR-2 mediates the accumulation of macrophages in atherosclerotic lesions of LDL receptor-deficient mice.

5. A new class of membrane-bound chemokine with a CX3C motif

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