Hormone Replacement Therapy and the Factor V Leiden Mutation
Author:
Affiliation:
1. From the Veterans’ Affairs Boston Healthcare System and Beth Israel Deaconess Medical Center, Harvard Medical School Boston, Mass.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine
Reference19 articles.
1. Familial thrombophilia due to a previously unrecognized mechanism characterized by poor anticoagulant response to activated protein C: prediction of a cofactor to activated protein C.
2. Mutation in blood coagulation factor V associated with resistance to activated protein C
3. Characterization of the Molecular Defect in Factor VR506A
4. Venous thrombosis due to poor anticoagulant response to activated protein C: Leiden Thrombophilia Study
5. High risk of thrombosis in patients homozygous for factor V Leiden (activated protein C resistance) [see comments]
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1. Conjugated Equine Estrogen, Esterified Estrogen, Prothrombotic Variants, and the Risk of Venous Thrombosis in Postmenopausal Women;Arteriosclerosis, Thrombosis, and Vascular Biology;2006-12
2. The use and abuse of screening tests around menopause;Climacteric;2006-01
3. Venous Thromboembolic Disease;Obstetrics & Gynecology;2004-10-01
4. Factor V Leiden and Venous Thromboembolism: Risk Associated With Hormone Replacement Therapy;Journal of the American Academy of Nurse Practitioners;2003-10
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