Tumor Necrosis Factor-α–Induced Iron Sequestration and Oxidative Stress in Human Endothelial Cells-Reference-Cited by-同舟云学术

Tumor Necrosis Factor-α–Induced Iron Sequestration and Oxidative Stress in Human Endothelial Cells

Published:2005-12 Issue:12 Volume:25 Page:2495-2501
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Nanami Masayoshi¹,Ookawara Tomomi¹,Otaki Yoshinaga¹,Ito Katsukiyo¹,Moriguchi Rintarou¹,Miyagawa Koji¹,Hasuike Yukiko¹,Izumi Masaaki¹,Eguchi Hironobu¹,Suzuki Keiichiro¹,Nakanishi Takeshi¹

Affiliation:

1. From the Department of Internal Medicine, Division of Kidney and Dialysis (M.N., Y.O., K.I., R.M., K.M., Y.K., M.I., T.N.) and the Department of Biochemistry (T.O., H.E., K.S.), Hyogo College of Medicine, Hyogo, Japan.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference37 articles.

1. The pathogenesis of atherosclerosis: a perspective for the 1990s

2. Elevated Circulating Levels of Tumor Necrosis Factor in Severe Chronic Heart Failure

3. Endothelial Function Testing as a Biomarker of Vascular Disease

4. Superoxide, H2O2, and iron are required for TNF-α-induced MCP-1 gene expression in endothelial cells: role of Rac1 and NADPH oxidase

5. Rac1 inhibits TNF‐α‐induced endothelial cell apoptosis: dual regulation by reactive oxygen species

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