Apolipoprotein B100 Metabolism in Autosomal-Dominant Hypercholesterolemia Related to Mutations inPCSK9-Reference-Cited by-同舟云学术

Apolipoprotein B100 Metabolism in Autosomal-Dominant Hypercholesterolemia Related to Mutations inPCSK9

Published:2004-08 Issue:8 Volume:24 Page:1448-1453
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Ouguerram Khadija¹,Chetiveaux Maud¹,Zair Yassine¹,Costet Philippe¹,Abifadel Marianne¹,Varret Mathilde¹,Boileau Catherine¹,Magot Thierry¹,Krempf Michel¹

Affiliation:

1. From INSERM U 539 (K.O., M.C., Y.Z., P.C., T.M., M.K.), Centre de Recherche en Nutrition Humaine de Nantes; and Hôtel Dieu (M.A., M.V., C.B.), Nantes, INSERM U 383, Hôpital Necker-enfants malades, Paris, France.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference38 articles.

1. Microsomal triglyceride transfer protein, the abetalipoproteinemia gene product, mediates the secretion of apolipoprotein B-containing lipoproteins from heterologous cells.

2. Association of coronary atherosclerosis with hyperapobetalipoproteinemia [increased protein but normal cholesterol levels in human plasma low density (beta) lipoproteins].

3. Apolipoprotein B metabolism in homozygous familial hypercholesterolemia.

4. Comparison of apolipoprotein B metabolism in familial defective apolipoprotein B and heterogeneous familial hypercholesterolemia

5. Ten LDL Receptor Mutants Explain One Third of Familial Hypercholesterolemia in a German Sample

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