Persistent Defect in Transmitter Release and Synapsin Phosphorylation in Cerebral Cortex After Transient Moderate Ischemic Injury-Reference-Cited by-同舟云学术

Persistent Defect in Transmitter Release and Synapsin Phosphorylation in Cerebral Cortex After Transient Moderate Ischemic Injury

Published:2002-05 Issue:5 Volume:33 Page:1369-1375
ISSN:0039-2499
Container-title:Stroke
language:en
Short-container-title:Stroke

Author:

Bolay Hayrunnisa¹,Gürsoy-Özdemir Yasemin¹,Sara Yildirim¹,Onur Rüstü¹,Can Alp¹,Dalkara Turgay¹

Affiliation:

1. From the Departments of Neurology (H.B., Y.G-Ö., T.D.) and Pharmacology (Y.S., R.O.), Faculty of Medicine, and Institute of Neurological Sciences and Psychiatry (H.B., Y.G-Ö., T.D.), Hacettepe University, and Department of Histology-Embryology, Ankara University School of Medicine (A.C.), Ankara, Turkey.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Advanced and Specialized Nursing,Cardiology and Cardiovascular Medicine,Neurology (clinical)

Reference43 articles.

1. Back T Nedergaard M Ginsberg MD. Cell signaling and ischemic neuronal death.In: Ginsberg MD Bogousslavsky J eds. Cerebrovascular Disease: Pathophysiology Diagnosis and Management. Oxford UK: Blackwell Science; 1998: 276–286.

2. Mechanisms involved in irreversible anoxic damage to thein vitrorat hippocampal slice

3. Ischemic Cell Death in Brain Neurons

4. Synapse Loss and Regeneration: A Mechanism for Functional Decline and Recovery after Cerebral Ischemia?

5. Effects of hypoxia on the monosynaptic reflex pathway in the cat spinal cord.

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