Angiotensin Receptor Blockade With Candesartan Attenuates Atherosclerosis, Plaque Disruption, and Macrophage Accumulation Within the Plaque in a Rabbit Model-Reference-Cited by-同舟云学术

Angiotensin Receptor Blockade With Candesartan Attenuates Atherosclerosis, Plaque Disruption, and Macrophage Accumulation Within the Plaque in a Rabbit Model

Published:2004-10-05 Issue:14 Volume:110 Page:2060-2065
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Johnstone Michael T.¹,Perez Alexandra S.¹,Nasser Imad¹,Stewart Robert¹,Vaidya Anand¹,Al Ammary Fawaz¹,Schmidt Ben¹,Horowitz Gary¹,Dolgoff Jennifer¹,Hamilton James¹,Quist William C.¹

Affiliation:

1. From the Departments of Medicine (Cardiovascular Division: M.T.J., A.S.P., A.V., F.A., B.S., J.D.), Surgery (R.S.), and Pathology (I.N., G.H., W.C.Q.), Beth Israel Deaconess Medical Center and Harvard Medical School, and the Department of Biophysics and Physiology (J.H.), Boston University School of Medicine, Boston, Mass.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference27 articles.

1. Introduction

2. Induction of platelet-derived growth factor A-chain and c-myc gene expressions by angiotensin II in cultured rat vascular smooth muscle cells.

3. Multiple autocrine growth factors modulate vascular smooth muscle cell growth response to angiotensin II.

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