Cardiac-Specific Overexpression of GLUT1 Prevents the Development of Heart Failure Attributable to Pressure Overload in Mice-Reference-Cited by-同舟云学术

Cardiac-Specific Overexpression of GLUT1 Prevents the Development of Heart Failure Attributable to Pressure Overload in Mice

Published:2002-10-15 Issue:16 Volume:106 Page:2125-2131
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Liao Ronglih¹,Jain Mohit¹,Cui Lei¹,D’Agostino Jessica¹,Aiello Francesco¹,Luptak Ivan¹,Ngoy Soeun¹,Mortensen Richard M.¹,Tian Rong¹

Affiliation:

1. From the Cardiac Muscle Research Laboratory, Whitaker Cardiovascular Institute, Department of Medicine (R.L., M.J., L.C., S.N.), Boston University School of Medicine, and NMR Laboratory for Physiological Chemistry, Division of Cardiovascular Medicine (R.L., J.D.A., F.A., I.L., R.T.), Brigham and Women’s Hospital and Harvard Medical School, Boston, Mass; and Department of Physiology and Medicine (R.M.M.), University of Michigan, Ann Arbor, Mich.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference30 articles.

1. Katz AM. Heart Failure: Pathophysiology Molecular Biology and Clinical Management. Philadelphia Pa: Lippincott Williams & Wilkins; 2000.

2. Increased glycolytic metabolism in cardiac hypertrophy and congestive failure

3. Insulin action on heart and skeletal muscle glucose uptake in essential hypertension.

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