Toll-Like Receptor 2 Mediates Staphylococcus aureus –Induced Myocardial Dysfunction and Cytokine Production in the Heart-Reference-Cited by-同舟云学术

Toll-Like Receptor 2 Mediates Staphylococcus aureus –Induced Myocardial Dysfunction and Cytokine Production in the Heart

Published:2004-12-14 Issue:24 Volume:110 Page:3693-3698
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Knuefermann Pascal¹,Sakata Yasushi¹,Baker J. Scott¹,Huang Chien-Hua¹,Sekiguchi Kenichi¹,Hardarson Hordur S.¹,Takeuchi Osamu¹,Akira Shizuo¹,Vallejo Jesus G.¹

Affiliation:

1. From the Department of Pediatrics (P.K., J.S.B., C.-H.H., H.S.H., J.G.V.), Section of Infectious Diseases, and the Winters Center for Heart Failure Research (P.K., Y.S., S.-H.H., K.S., J.G.V.), Baylor College of Medicine and Texas Children’s Hospital, Houston, Tex, and the Department of Host Defense (O.T., S.A.), Osaka University, Osaka, Japan.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference26 articles.

1. Profound but Reversible Myocardial Depression in Patients with Septic Shock

2. Role of endotoxemia in cardiovascular dysfunction and mortality. Escherichia coli and Staphylococcus aureus challenges in a canine model of human septic shock.

3. Left ventricular diastolic function in sepsis

4. Persistent Preload Defect in Severe Sepsis Despite Fluid Loading

5. The tissue distribution of tumor necrosis factor biosynthesis during endotoxemia.

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