Level of β-Adrenergic Receptor Kinase 1 Inhibition Determines Degree of Cardiac Dysfunction After Chronic Pressure Overload–Induced Heart Failure-Reference-Cited by-同舟云学术

Level of β-Adrenergic Receptor Kinase 1 Inhibition Determines Degree of Cardiac Dysfunction After Chronic Pressure Overload–Induced Heart Failure

Published:2005-02-08 Issue:5 Volume:111 Page:591-597
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Tachibana Hideo¹,Naga Prasad Sathyamangla V.¹,Lefkowitz Robert J.¹,Koch Walter J.¹,Rockman Howard A.¹

Affiliation:

1. From the Departments of Medicine (H.T., S.V.N.P., R.J.L., H.A.R.), Cell Biology (H.A.R.), Molecular Genetics (H.A.R.), and Surgery (W.J.K.), and the Howard Hughes Medical Institute (R.J.L.), Duke University Medical Center, Durham, NC. Dr Koch is now at the Center for Translational Medicine, Jefferson Medical College, Philadelphia, Pa.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference33 articles.

1. Beta-adrenergic neuroeffector abnormalities in the failing human heart are produced by local rather than systemic mechanisms.

2. Inhibition of receptor-localized PI3K preserves cardiac β-adrenergic receptor function and ameliorates pressure overload heart failure

3. Seven-transmembrane-spanning receptors and heart function

4. G protein—coupled receptor kinases

5. G Protein-coupled Receptors

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