Periostin as a Novel Factor Responsible for Ventricular Dilation-Reference-Cited by-同舟云学术

Periostin as a Novel Factor Responsible for Ventricular Dilation

Published:2004-09-28 Issue:13 Volume:110 Page:1806-1813
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Katsuragi Naruto¹,Morishita Ryuichi¹,Nakamura Noriko¹,Ochiai Tayehito¹,Taniyama Yoshiaki¹,Hasegawa Yasuhiro¹,Kawashima Kayoko¹,Kaneda Yasufumi¹,Ogihara Toshio¹,Sugimura Keijiro¹

Affiliation:

1. From Daiichi Suntory Biomedical Research Ltd (N.K., N.N., T. Ochiai, Y.H., K.K., K.S.), Osaka; the Divisions of Clinical Gene Therapy (R.M., Y.T.) and of Gene Therapy Science (Y.K.), and the Department of Geriatric Medicine (T. Ogihara), Osaka University Graduate School of Medicine, Suita, Japan.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference32 articles.

1. Effect of Enalapril on Mortality and the Development of Heart Failure in Asymptomatic Patients with Reduced Left Ventricular Ejection Fractions

2. A molecular basis for familial hypertrophic cardiomyopathy: A β cardiac myosin heavy chain gene missense mutation

3. α-tropomyosin and cardiac troponin T mutations cause familial hypertrophic cardiomyopathy: A disease of the sarcomere

4. Mutations in the cardiac myosin binding protein–C gene on chromosome 11 cause familial hypertrophic cardiomyopathy

5. Cardiac myosin binding protein–C gene splice acceptor site mutation is associated with familial hypertrophic cardiomyopathy

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