Alterations in Janus Kinase (JAK)-Signal Transducers and Activators of Transcription (STAT) Signaling in Patients With End-Stage Dilated Cardiomyopathy-Reference-Cited by-同舟云学术

Alterations in Janus Kinase (JAK)-Signal Transducers and Activators of Transcription (STAT) Signaling in Patients With End-Stage Dilated Cardiomyopathy

Published:2003-02-18 Issue:6 Volume:107 Page:798-802
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Podewski Edith K.¹,Hilfiker-Kleiner Denise¹,Hilfiker Andres¹,Morawietz Henning¹,Lichtenberg Artur¹,Wollert Kai C.¹,Drexler Helmut¹

Affiliation:

1. From the Departments of Cardiology and Angiology (E.K.P., D.H.K., A.H., K.C.W., H.D.), and Cardiovascular Surgery (A.L.), Hannover Medical School, Hannover, Germany, and the Institute of Pathophysiology (H.M.), University of Halle-Wittenberg, Germany.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference17 articles.

1. Signaling Pathways for Cardiac Hypertrophy and Failure

2. Loss of a gp130 Cardiac Muscle Cell Survival Pathway Is a Critical Event in the Onset of Heart Failure during Biomechanical Stress

3. Cardiotrophin 1 (CT-1) Inhibition of Cardiac Myocyte Apoptosis via a Mitogen-activated Protein Kinase-dependent Pathway

4. Signal transducer and activator of transcription 3 in the heart transduces not only a hypertrophic signal but a protective signal against doxorubicin-induced cardiomyopathy

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