Engagement of Glycoprotein IIb/IIIa (αIIbβ3) on Platelets Upregulates CD40L and Triggers CD40L-Dependent Matrix Degradation by Endothelial Cells-Reference-Cited by-同舟云学术

Engagement of Glycoprotein IIb/IIIa (αIIbβ3) on Platelets Upregulates CD40L and Triggers CD40L-Dependent Matrix Degradation by Endothelial Cells

Published:2002-10-15 Issue:16 Volume:106 Page:2111-2117
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

May Andreas E.¹,Kälsch Thorsten¹,Massberg Steffen¹,Herouy Yared¹,Schmidt Roland¹,Gawaz Meinrad¹

Affiliation:

1. From Medizinische Klinik, Klinikum rechts der Isar and Deutsches Herzzentrum, Technische Universität München, and Department of Dermatology, University Medical School (Y.H.), Freiburg, Germany. Dr Kälsch is now at Medizinische Klinik, Universitätsklinikum Mannheim, Germany.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference24 articles.

1. Inhibition of plasminogen activators or matrix metalloproteinases prevents cardiac rupture but impairs therapeutic angiogenesis and causes cardiac failure

2. Increased expression of matrix metalloproteinases and matrix degrading activity in vulnerable regions of human atherosclerotic plaques.

3. Urokinase receptor: a molecular organizer in cellular communication

4. Tissue cooperation in a proteolytic cascade activating human interstitial collagenase.

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