PRIMPOL competes with RAD51 to resolve G-quadruplex-induced replication stress via its interaction with RPA
Author:
Publisher
China Science Publishing & Media Ltd.
Subject
General Medicine,Biochemistry,Biophysics
Link
https://engine.scichina.com/doi/pdf/51F9974E19324A75BD58E9F9317A2253
Reference40 articles.
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2. Quinet A, Lemaçon D, Vindigni A. Replication fork reversal: players and guardians. Mol Cell, 2017, 68: 830-833.
3. Batista LF, Kaina B, Meneghini R, Menck CF. How DNA lesions are turned into powerful killing structures: insights from UV-induced apoptosis. Mutat Res Rev Mutat Res, 2009, 681: 197-208.
4. Frasson I, Pirota V, Richter SN, Doria F. Multimeric G-quadruplexes: a review on their biological roles and targeting. Int J Biol Macromolecules, 2022, 204: 89-102.
5. Panichnantakul P, Patel A, Tse EY, Wyatt HD. An open-source platform to quantify subnuclear foci and protein colocalization in response to replication stress. DNA Repair, 2021, 105:
Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. PrimPol variant V102A with altered primase and polymerase activities;2023-11-29
2. G-quadruplex resolution: From molecular mechanisms to physiological relevance;DNA Repair;2023-10
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