Endoplasmic reticulum stress caused by traumatic injury promotes cardiomyocyte apoptosis through acetylation modification of GRP78-Reference-Cited by-同舟云学术

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Endoplasmic reticulum stress caused by traumatic injury promotes cardiomyocyte apoptosis through acetylation modification of GRP78

Published:2023-12-15 Issue:1 Volume:56 Page:96-105
ISSN:1672-9145
Container-title:Acta Biochimica et Biophysica Sinica
language:en
Short-container-title:ABBS

Author:

Yan Zi,Liu Yufeng,Yang Bowen,Zhao Wenhui,Wang Yan,Wang Deping,Li Jianguo,Jiao Xiangying,Cao Jimin

Publisher

China Science Publishing & Media Ltd.

Subject

General Medicine,Biochemistry,Biophysics,Molecular Biology

Link

https://engine.scichina.com/doi/pdf/241E0FD584474127B5D29E45447FF515

Reference38 articles.

1. Weber B, Lackner I, Gebhard F, Miclau T, Kalbitz M. Trauma, a matter of the heart—molecular mechanism of post-traumatic cardiac dysfunction. Int J Mol Sci, 2021, 22:

2. De′Ath HD, Manson J, Davenport R, Glasgow S, Renfrew I, Davies LC, Uppal R. Trauma-induced secondary cardiac injury is associated with hyperacute elevations in inflammatory cytokines. Shock, 2013, 39: 415-420.

3. De′Ath HD, Rourke C, Davenport R, Manson J, Renfrew I, Uppal R, Davies LC. Clinical and biomarker profile of trauma-induced secondary cardiac injury. Br J Surg, 2012, 99: 789-797.

4. Schmidt U, Oramary D, Kamin K, Buschmann CT, Kleber C. Synergistic effects of forensic medicine and traumatology: comparison of clinical diagnosis autopsy findings in trauma-related deaths. World J Surg, 2020, 44: 1137-1148.

5. Wall J, Naganathar S, Praditsuktavorn B, Bugg OF, McArthur S, Thiemermann C, Tremoleda JL. Modeling cardiac dysfunction following traumatic hemorrhage injury: impact on myocardial integrity. Front Immunol, 2019, 10:

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