Forsythiaside Protected H9c2 Cardiomyocytes from H<sub>2</sub>O<sub>2</sub>-Induced Oxidative Stress and Apoptosis <i>via</i> Activating Nrf2/HO-1 Signaling Pathway
Author:
Affiliation:
1. Department of Cardiology, The Affiliated Hospital of Shaanxi University of Traditional Chinese Medicine
Publisher
International Heart Journal (Japanese Heart Journal)
Subject
Cardiology and Cardiovascular Medicine,General Medicine
Link
https://www.jstage.jst.go.jp/article/ihj/63/5/63_21-585/_pdf
Reference47 articles.
1. 1. Birben E, Sahiner UM, Sackesen C, Erzurum S, Kalayci O. Oxidative stress and antioxidant defense. World Allergy Organ J 2012; 5: 9-19.
2. 2. Shinjo T, Tanaka T, Okuda H, et al. Propofol induces nuclear localization of Nrf2 under conditions of oxidative stress in cardiac H9c2 cells. PLoS One 2018; 13: e0196191.
3. 3. Ding B, Abe JI, Wei H, et al. Functional role of phosphodiesterase 3 in cardiomyocyte apoptosis: implication in heart failure. Circulation 2005; 111: 2469-76.
4. 4. Hutcheson R, Rocic P. The metabolic syndrome, oxidative stress, environment, and cardiovascular disease: the great exploration. Exp Diabetes Res 2012; 2012: 271028.
5. 5. Laflamme MA, Chen KY, Naumova AV, et al. Cardiomyocytes derived from human embryonic stem cells in pro-survival factors enhance function of infarcted rat hearts. Nat Biotechnol 2007; 25: 1015-24.
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