The Suppression of Proinflammatory Cytokines Improves Heart Function From Non-Heart-Beating Donors Following Transplantation in a Canine Model
Author:
Affiliation:
1. Department of Thoracic and Visceral Organ Surgery, Gunma University Graduate School of Medicine
2. Intensive Care Unit, Gunma University Hospital
3. CML Pathological Research
Publisher
International Heart Journal (Japanese Heart Journal)
Subject
Cardiology and Cardiovascular Medicine,General Medicine
Link
http://www.jstage.jst.go.jp/article/ihj/50/2/50_2_235/_pdf
Reference33 articles.
1. 1. Scheule AM, Jost D, Beierlein W, et al. Sodium-hydrogen inhibitor cariporide (HOE642) improves in situ protection of heart from non-heart-beating donors. J Heart Lung Transplant 2003; 22: 1335-42.
2. 2. Wuerflein RD, Shumway NE. Resuscitation and function of the cadaver heart. Circulation 1967; 35: I92-5.
3. 3. Bretschneider HJ, Hüebner G, Knoll D, Lohr D, Nordbeck H, Spieckermann PG. Myocardial resistance and tolerance to ischemia: physiological and biochemical basis. J Cardiovasc Surg 1975; 16: 241-60. (Review)
4. 4. Suehiro K, Mohri M, Yamaguchi H, et al. Posttransplant function of a nonbeating heart is predictable by an ex vivo perfusion method. Ann Thorac Surg 2001; 71: 278-83.
5. Effect of FR167653, a cytokine suppressive agent, on endotoxin-induced disseminated intravascular coagulation
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