Norepinephrine-induced Inositol 1,4,5-Trisphosphate Formation in Atrial Myocytes is Regulated by Extracellular Calcium, Protein Kinase C, and Calmodulin
Author:
Affiliation:
1. Department of Anesthesiology, Hirosaki National Hospital
2. Department of Pediatrics, Hirosaki National Hospital
Publisher
International Heart Journal (Japanese Heart Journal)
Subject
Cardiology and Cardiovascular Medicine
Link
https://www.jstage.jst.go.jp/article/jhj/44/4/44_4_547/_pdf
Reference18 articles.
1. 1. Ambler SK, Leite MF. Regulation of atrial natriuretic peptide secretion by alpha1-adrenergic receptors: the role of a different second messenger pathway. J Mol Cell Cardiol 1994; 26: 391-402.
2. 2. Brunner F, Mouton R, Opie LH. Defective stretch-induced release of atrial natriuretic peptide from aging hypertensive rat heart: possible role of phosphatidylinositol pathway. J Cardiovasc Pharmacol 1995; 25: 168-76.
3. 3. Hayashi H, Miyata H, Terada H, et al. Effects of phospholipase C on action potentials and intracellular Ca2+ concentration in guinea pig heart. Jpn Circ J 1993; 57: 344-52.
4. 4. Endo T, Naka M, Hidaka H. Ca2+ phospholipid-dependent phosphorylation of smooth muscle myosin. Biochem Biophys Res Comm 1982; 105: 942-8.
5. 5. Brooks WW, Conrad CH, Morgan JP. Reperfusion-induced arrhythmias following ischemia in intact rat heart: role of intracellular calcium. Cardiovas Res 1995; 29: 536-42.
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