Cardiac Muscle Cell Disorganization in Apical Hypertrophic Cardiomyopathy
Author:
Affiliation:
1. Division of Cardiology, Department of Internal Medicine, Fujita Health University School of Medicine, Toyoake
2. Department of Internal Medicine, Heart Institute of Japan, Tokyo Women's Medical University
Publisher
International Heart Journal (Japanese Heart Journal)
Subject
Cardiology and Cardiovascular Medicine
Link
http://www.jstage.jst.go.jp/article/jhj/44/4/44_4_505/_pdf
Reference28 articles.
1. A molecular basis for familial hypertrophic cardiomyopathy: A β cardiac myosin heavy chain gene missense mutation
2. A molecular basis for familial hypertrophic cardiomyopathy: An αβ cardiac myosin heavy chain hybrid gene
3. α-tropomyosin and cardiac troponin T mutations cause familial hypertrophic cardiomyopathy: A disease of the sarcomere
4. Codon 102 of the Cardiac Troponin T Gene Is a Putative Hot Spot for Mutations in Familial Hypertrophic Cardiomyopathy
5. Patients With Familial Hypertrophic Cardiomyopathy Caused by a Phe110Ile Missense Mutation in the Cardiac Troponin T Gene Have Variable Cardiac Morphologies and a Favorable Prognosis
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