Angiotensin II Increases HMGB1 Expression in the Myocardium Through AT1 and AT2 Receptors When Under Pressure Overload
Author:
Affiliation:
1. Department of Cardiology, Zhongshan Hospital, Fudan University
2. Shanghai Institute of Cardiovascular Diseases
3. Department of General Practice, Zhongshan Hospital, Fudan University
Publisher
International Heart Journal (Japanese Heart Journal)
Subject
Cardiology and Cardiovascular Medicine,General Medicine
Link
https://www.jstage.jst.go.jp/article/ihj/62/1/62_20-384/_pdf
Reference44 articles.
1. 1. Kumar S, Wang G, Zheng N, et al. HIMF (hypoxia-induced mitogenic factor) -IL (interleukin) -6 signaling mediates cardiomyocyte-fibroblast crosstalk to promote cardiac hypertrophy and fibrosis. Hypertension 2019; 73: 1058-70.
2. 2. Zablocki D, Sadoshima J. Solving the cardiac hypertrophy riddle: The angiotensin II-mechanical stress connection. Circ Res 2013; 113: 1192-5.
3. 3. Matsushita N, Ishida N, Ibi M, et al. Chronic pressure overload induces cardiac hypertrophy and fibrosis via increases in SGLT1 and IL-18 gene expression in mice. Int Heart J 2018; 59: 1123-33.
4. 4. Blaustein MP. How does pressure overload cause cardiac hypertrophy and dysfunction? High-ouabain affinity cardiac Na+ pumps are crucial. Am J Physiol Heart Circ Physiol 2017; 313: H919-30.
5. 5. Gesmundo I, Miragoli M, Carullo P, et al. Growth hormone-releasing hormone attenuates cardiac hypertrophy and improves heart function in pressure overload-induced heart failure. Proc Natl Acad Sci U S A 2017; 114: 12033-8.
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