Potential Effects of Ischemic Postconditioning and Changes in Heat Shock Protein 72 in Patients with Acute Myocardial Infarction without Prodromal Angina-Reference-Cited by-同舟云学术

Potential Effects of Ischemic Postconditioning and Changes in Heat Shock Protein 72 in Patients with Acute Myocardial Infarction without Prodromal Angina

Published:2024-05-31 Issue:3 Volume:65 Page:395-403
ISSN:1349-2365
Container-title:International Heart Journal
language:en
Short-container-title:Int. Heart J.

Author:

Takeuchi Toshiharu¹,Kitani Yuya¹,Minoshima Akiho¹,Ota Hisanobu¹,Nakagawa Naoki¹,Sumitomo Kazuhiro²,Ishii Yoshinao³,Hasebe Naoyuki¹

Affiliation:

1. Division of Cardiology, Department of Internal Medicine, Asahikawa Medical University

2. Department of Community Medicine, Tohoku Medical and Pharmaceutical University Wakabayashi Hospital

3. Division of Cardiology, Department of Internal Medicine, Asahikawa City Hospital

Publisher

International Heart Journal (Japanese Heart Journal)

Link

https://www.jstage.jst.go.jp/article/ihj/65/3/65_23-651/_pdf

Reference36 articles.

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3. 3. Okada M, Hasebe N, Aizawa Y, Izawa K, Kawabe J, Kikuchi K. Thermal treatment attenuates neointimal thickening with enhanced expression of heat-shock protein 72 and suppression of oxidative stress. Circulation 2004; 109: 1763-8.

4. 4. Hampton CR, Shimamoto A, Rothnie CL, et al. HSP70.1 and -70.3 are required for late-phase protection induced by ischemic preconditioning of mouse hearts. Am J Physiol Heart Circ Physiol 2003; 285: H866-74.

5. 5. Tranter M, Ren X, Forde T, et al. NF-kappaB driven cardioprotective gene programs; Hsp70.3 and cardioprotection after late ischemic preconditioning. J Mol Cell Cardiol 2010; 49: 664-72.