Potential of Receptor for Advanced Glycation End-Products (RAGE) as an Eligible Biomarker for Therapy Evaluation in Patients With Pulmonary Hypertension
Author:
Affiliation:
1. Department of Cardiovascular Medicine, Graduate School of Medicine, The University of Tokyo
Publisher
International Heart Journal (Japanese Heart Journal)
Subject
Cardiology and Cardiovascular Medicine,General Medicine
Link
https://www.jstage.jst.go.jp/article/ihj/57/2/57_16-073/_pdf
Reference10 articles.
1. 1. Abdul-Salam VB, Wharton J, Cupitt J, Berryman M, Edwards RJ, Wilkins MR. Proteomic analysis of lung tissues from patients with pulmonary arterial hypertension. Circulation 2010; 122: 2058-67.
2. 2. Meloche J, Courchesne A, Barrier M, et al. Critical role for the advanced glycation end-products receptor in pulmonary arterial hypertension etiology. J Am Heart Assoc 2013; 2: e005157.
3. 3. Bauer EM, Shapiro R, Zheng H, et al. High mobility group box 1 contributes to the pathogenesis of experimental pulmonary hypertension via activation of Toll-like receptor 4. Mol Med 2013; 18: 1509-18.
4. 4. Sadamura-Takenaka Y, Ito T, Noma S, et al. HMGB1 promotes the development of pulmonary arterial hypertension in rats. PLoS One 2014; 9: e102482.
5. 5. Farmer DG, Ewart MA, Mair KM, Kennedy S. Soluble receptor for advanced glycation end products (sRAGE) attenuates haemodynamic changes to chronic hypoxia in the mouse. Pulm Pharmacol Ther 2014; 29: 7-14.
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2. Impairment of Iodine-123-Metaiodobenzylguanidine (123I-MIBG) Uptake in Patients with Pulmonary Artery Hypertension;International Heart Journal;2018
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