MiR-324-3p Regulates Fibroblast Proliferation via Targeting TGF-β1 in Atrial Fibrillation-Reference-Cited by-同舟云学术

MiR-324-3p Regulates Fibroblast Proliferation via Targeting TGF-β1 in Atrial Fibrillation

Published:2020-11-28 Issue:6 Volume:61 Page:1270-1278
ISSN:1349-2365
Container-title:International Heart Journal
language:en
Short-container-title:Int. Heart J.

Author:

Xu Jiali¹,Lei Sisi¹,Sun Shuo²,Zhang Wei²,Zhu Feiyu²³,Yang Haichen²,Xu Qingmei²³,Zhang Bing²,Li Hui²,Zhu Mingli²,Hu Xiangwen²,Zhang Heng²,Tang Bi²,Kang Pinfang²³

Affiliation:

1. Department of Pediatrics, The First Affiliated Hospital of Bengbu Medical College

2. Department of Cardiovascular Disease, The First Affiliated Hospital of Bengbu Medical College

3. Center for Cardiovascular and Cerebrovascular Research, Bengbu Medical College

Publisher

International Heart Journal (Japanese Heart Journal)

Subject

Cardiology and Cardiovascular Medicine,General Medicine

Link

https://www.jstage.jst.go.jp/article/ihj/61/6/61_20-423/_pdf

Reference40 articles.

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2. 2. Kumar P, Kiser AC, Gehi AK. Hybrid treatment of atrial fibrillation. Prog Cardiovasc Dis 2015; 58: 213-20.

3. 3. Margulescu AD, Mont L. Persistent atrial fibrillation vs paroxysmal atrial fibrillation: differences in management. Expert Rev Cardiovasc Ther 2017; 15: 601-18.

4. 4. Nattel S. Molecular and cellular mechanisms of atrial fibrosis in atrial fibrillation. JACC Clin Electrophysiol 2017; 3: 425-35.

5. 5. Yoo S, Aistrup G, Shiferaw Y, et al. Oxidative stress creates a unique, CaMKII-mediated substrate for atrial fibrillation in heart failure. JCI Insight 2018; 3.

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