The Agonist of Inward Rectifier Potassium Channel (I<sub>K1</sub>) Attenuates Rat Reperfusion Arrhythmias Linked to CaMKII Signaling-Reference-Cited by-同舟云学术

The Agonist of Inward Rectifier Potassium Channel (I_K1) Attenuates Rat Reperfusion Arrhythmias Linked to CaMKII Signaling

Published:2021-11-29 Issue:6 Volume:62 Page:1348-1357
ISSN:1349-2365
Container-title:International Heart Journal
language:en
Short-container-title:Int. Heart J.

Author:

Liu Qinghua¹,Sun Jiaxing¹,Zhang Lijun¹,Xu Yanwu²,Wu Bowei³,Cao Jimin³

Affiliation:

1. Department of Pathophysiology, Shanxi Medical University

2. Department of Biochemistry, Shanghai University of Traditional Chinese Medicine

3. Key Laboratory of Cellular Physiology at Shanxi Medical University, Ministry of Education, and the Department of Physiology, Shanxi Medical University

Publisher

International Heart Journal (Japanese Heart Journal)

Subject

Cardiology and Cardiovascular Medicine,General Medicine

Link

https://www.jstage.jst.go.jp/article/ihj/62/6/62_21-379/_pdf

Reference38 articles.

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2. 2. Strömer H, de Groot MC, Horn M, et al. Na (+) /H (+) exchange inhibition with HOE642 improves postischemic recovery due to attenuation of Ca (2+) overload and prolonged acidosis on reperfusion. Circulation 2000; 101: 2749-55.

3. 3. Inserte J, Barrabés JA, Hernando V, Garcia-Dorado D. Orphan targets for reperfusion injury. Cardiovasc Res 2009; 83: 169-78.

4. 4. Sedlis SP. Mechanisms of ventricular arrhythmias in acute ischemia and reperfusion. Cardiovasc Clin 1992; 22: 3-18.

5. 5. Zweier JL. Measurement of superoxide-derived free radicals in the reperfused heart. Evidence for a free radical mechanism of reperfusion injury. J Biol Chem 1988; 263: 1353-7.

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