NK Cells Contribute to the Control of Trypanosoma cruzi Infection by Killing Free Parasites by Perforin-Independent Mechanisms
Author:
Affiliation:
1. Department of Immunology, Bernhard Nocht Institute for Tropical Medicine
2. Institute of Immunology, University Hospital, Hamburg, Germany
Abstract
Publisher
American Society for Microbiology
Subject
Infectious Diseases,Immunology,Microbiology,Parasitology
Link
https://journals.asm.org/doi/pdf/10.1128/IAI.72.12.6817-6825.2004
Reference35 articles.
1. Interleukin-12 mediates resistance to Trypanosoma cruzi in mice and is produced by murine macrophages in response to live trypomastigotes
2. Antunez, M. I., and R. L. Cardoni. 2000. IL-12 and IFN-γ production, and NK cell activity, in acute and chronic experimental Trypanosoma cruzi infections. Immunol. Lett.71:103-109.
3. Bisaggio Rda, C., S. L. de Castro, H. S. Barbosa, A. Brandao Cde, and P. M. Persechini. 1997. Trypanosoma cruzi: resistance to the pore forming protein of cytotoxic lymphocytes—perforin. Exp. Parasitol.86:144-154.
4. Bustin, S. A. 2000. Absolute quantification of mRNA using real-time reverse transcription polymerase chain reaction assays. J. Mol. Endocrinol.25:169-193.
5. Cardillo, F., F. Q. Cunha, W. M. Tamashiro, M. Russo, S. B. Garcia, and J. Mengel. 2002. NK1.1+ cells and T-cell activation in euthymic and thymectomized C57BL/6 mice during acute Trypanosoma cruzi infection. Scand. J. Immunol.55:96-104.
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