Bif-1c Attenuates Viral Proliferation by Regulating Autophagic Flux Blockade Induced by the Rabies Virus CVS-11 Strain in N2a Cells

Author:

Hou Pengfei12,Guo Yidi1,Jin Hongli12,Sun Jingxuan1,Bai Yujie1,Li Wujian12,Li Ling3,Cao Zengguo12,Wu Fangfang2,Zhang Haili1,Li Yuanyuan1,Yang Songtao2,Xia Xianzhu2,Huang Pei1,Wang Hualei1ORCID

Affiliation:

1. Key Laboratory of Zoonosis Research, Ministry of Education, College of Veterinary Medicine, Jilin University, Changchun, China

2. Changchun Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Changchun, China

3. National Research Center for Exotic Animal Diseases, China Animal Health and Epidemiology Center, Qingdao, China

Abstract

Autophagy can be triggered by viral infection and replication. Autophagosomes are generated and affect RABV replication, which differs by viral strain and infected cell type.

Funder

National Natural Science Foundation of China

Publisher

American Society for Microbiology

Subject

Infectious Diseases,Cell Biology,Microbiology (medical),Genetics,General Immunology and Microbiology,Ecology,Physiology

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