Bif-1c Attenuates Viral Proliferation by Regulating Autophagic Flux Blockade Induced by the Rabies Virus CVS-11 Strain in N2a Cells-Reference-Cited by-同舟云学术

Bif-1c Attenuates Viral Proliferation by Regulating Autophagic Flux Blockade Induced by the Rabies Virus CVS-11 Strain in N2a Cells

Published:2023-06-15 Issue:3 Volume:11 Page:
ISSN:2165-0497
Container-title:Microbiology Spectrum
language:en
Short-container-title:Microbiol Spectr

Author:

Hou Pengfei¹²,Guo Yidi¹,Jin Hongli¹²,Sun Jingxuan¹,Bai Yujie¹,Li Wujian¹²,Li Ling³,Cao Zengguo¹²,Wu Fangfang²,Zhang Haili¹,Li Yuanyuan¹,Yang Songtao²,Xia Xianzhu²,Huang Pei¹,Wang Hualei¹^ORCID

Affiliation:

1. Key Laboratory of Zoonosis Research, Ministry of Education, College of Veterinary Medicine, Jilin University, Changchun, China

2. Changchun Veterinary Research Institute, Chinese Academy of Agricultural Sciences, Changchun, China

3. National Research Center for Exotic Animal Diseases, China Animal Health and Epidemiology Center, Qingdao, China

Abstract

Autophagy can be triggered by viral infection and replication. Autophagosomes are generated and affect RABV replication, which differs by viral strain and infected cell type.

Funder

National Natural Science Foundation of China

Publisher

American Society for Microbiology

Subject

Infectious Diseases,Cell Biology,Microbiology (medical),Genetics,General Immunology and Microbiology,Ecology,Physiology

Link

https://journals.asm.org/doi/pdf/10.1128/spectrum.03079-22

Reference33 articles.

1. The cell biology of rabies virus: using stealth to reach the brain

2. Human rabies: neuropathogenesis, diagnosis, and management

3. Rabies

4. The spread and evolution of rabies virus: conquering new frontiers

5. Autophagy: process and function

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