Defective Dendrite Elongation but Normal Fertility in Mice Lacking the Rho-Like GTPase Activator Dbl
Author:
Affiliation:
1. Dipartimento di Genetica, Biologia e Biochimica
2. Dipartimento di Anatomia, Farmacologia e Medicina Forense, Università di Torino, 10126 Turin
3. Laboratorio di Biologia Molecolare, Istituto G. Gaslini, 16147 Genoa, Italy
Abstract
Publisher
American Society for Microbiology
Subject
Cell Biology,Molecular Biology
Link
https://journals.asm.org/doi/pdf/10.1128/MCB.22.9.3140-3148.2002
Reference45 articles.
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3. Cherfils, J., and P. Chardin. 1999. GEFs: structural basis for their activation of small GTP-binding proteins. Trends Biochem. Sci. 24 : 306-311.
4. Colucci-D'Amato, G. L., G. Santelli, A. D'Alessio, G. Chiappetta, A. Mineo, G. Manzo, G. Vecchio, and V. de Franciscis. 1995. Dbl expression driven by the neuron specific enolase promoter induces tumor formation in transgenic mice with a p53(+/−) genetic background. Biochem. Biophys. Res. Commun. 216 : 762-770.
5. Coso, O. A., M. Chiariello, J. C. Yu, H. Teramoto, P. Crespo, N. Xu, T. Miki, and J. S. Gutkind. 1995. The small GTP-binding proteins Rac1 and Cdc42 regulate the activity of the JNK/SAPK signaling pathway. Cell 81 : 1137-1146.
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