Overproduction of transposon Tn10-encoded tetracycline resistance protein results in cell death and loss of membrane potential

Author:

Eckert B1,Beck C F1

Affiliation:

1. Institut für Biologie III, Albert-Ludwigs-Universität Freiburg, Federal Republic of Germany.

Abstract

High-level expression of the Tn10 tetracycline resistance protein TetA in Escherichia coli caused partial collapse of the membrane potential, arrest of growth, and killing of the cells. Since alpha-methylglucoside transport was not affected, the overproduced TetA protein may cause not destruction of membrane structure but rather unrestricted translocation of protons and/or ions across the membrane.

Publisher

American Society for Microbiology

Subject

Molecular Biology,Microbiology

Reference22 articles.

1. Tetracycline diffusion through phospholipid bilayers and binding to phospholipids;Argast M.;Antimicrob. Agents Chemother.,1984

2. Tetracycline uptake by susceptible Escherichia coli cells;Argast M.;Arch. Microbiol.,1985

3. A multifunctional gene (tetR) controls TnIO-encoded tetracycline resistance;Beck C. F.;J. Bacteriol.,1982

4. Overlapping divergent promoters control expression of TnIO tetracycline resistance;Bertrand K. P.;Gene,1983

5. Reduced expression of TnlO-mediated tetracycline resistance in Escherichia coli containing more than one copy of the transposon;Chopra I.;J. Gen. Microbiol.,1981

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