A Kaposi's Sarcoma-Associated Herpesvirus Infection Mechanism Is Independent of Integrins α3β1, αVβ3, and αVβ5-Reference-Cited by-同舟云学术

A Kaposi's Sarcoma-Associated Herpesvirus Infection Mechanism Is Independent of Integrins α3β1, αVβ3, and αVβ5

Published:2018-09 Issue:17 Volume:92 Page:
ISSN:0022-538X
Container-title:Journal of Virology
language:en
Short-container-title:J Virol

Author:

TerBush Allison Alwan¹,Hafkamp Florianne²,Lee Hee Jun¹,Coscoy Laurent¹

Affiliation:

1. Department of Molecular and Cell Biology, University of California, Berkeley, Berkeley, California, USA

2. Graduate School of Life Sciences, Utrecht University, Utrecht, Netherlands

Abstract

Our data reveal an integrin-independent route of KSHV infection and suggest that multiple Eph receptors besides EphA2 can promote and regulate infection. Since integrins and Eph receptors are large protein families with diverse expression patterns across cells and tissues, we propose that KSHV may engage with several proteins from both families in different combinations to negotiate successful entry into diverse cell types.

Publisher

American Society for Microbiology

Subject

Virology,Insect Science,Immunology,Microbiology

Link

https://journals.asm.org/doi/pdf/10.1128/JVI.00803-18

Reference72 articles.

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3. Host Range of Kaposi's Sarcoma-Associated Herpesvirus in Cultured Cells

4. KSHV Entry and Trafficking in Target Cells—Hijacking of Cell Signal Pathways, Actin and Membrane Dynamics

5. Monocytes in Kaposi's sarcoma lesions are productively infected by human herpesvirus 8

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