BonA from Acinetobacter baumannii Forms a Divisome-Localized Decamer That Supports Outer Envelope Function

Author:

Grinter Rhys1ORCID,Morris Faye C.1ORCID,Dunstan Rhys A.1ORCID,Leung Pok Man1ORCID,Kropp Ashleigh1,Belousoff Matthew12,Gunasinghe Sachith D.13,Scott Nichollas E.4ORCID,Beckham Simone5,Peleg Anton Y.16,Greening Chris1ORCID,Li Jian1ORCID,Heinz Eva17ORCID,Lithgow Trevor1ORCID

Affiliation:

1. Infection and Immunity Program, Biomedicine Discovery Institute and Department of Microbiology, Monash University, Clayton, Australia

2. Drug and Development Biology, Monash Institute of Pharmaceutical Sciences, Monash University, Parkville, Australia

3. EMBL Australia Node in Single Molecule Science, University of New South Wales, Sydney, Australia

4. Department of Microbiology and Immunology, University of Melbourne at the Peter Doherty Institute for Infection and Immunity, Melbourne, Australia

5. La Trobe Rural Health School, College of Science, Health and Engineering, La Trobe University, Bendigo, Australia

6. Department of Infectious Diseases, The Alfred Hospital and Central Clinical School, Monash University, Melbourne, Australia

7. Liverpool School of Tropical Medicine, Liverpool, United Kingdom

Abstract

The pathogen Acinetobacter baumannii is considered an urgent threat to human health. A. baumannii is highly resistant to treatment with antibiotics, in part due to its protective cell envelope. This bacterium is only distantly related to other bacterial pathogens, so its cell envelope has distinct properties and contains components distinct from those of other bacteria that support its function.

Funder

Wellcome

Department of Health, Australian Government | National Health and Medical Research Council

Publisher

American Society for Microbiology

Subject

Virology,Microbiology

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