Severe Schistosomiasis in the Absence of Interleukin-4 (IL-4) Is IL-12 Independent
Author:
Affiliation:
1. Department of Microbiology and Immunology, College of Veterinary Medicine, Cornell University, Ithaca, New York,1 and
2. Basel Institute for Immunology, Basel, Switzerland2
Abstract
Publisher
American Society for Microbiology
Subject
Infectious Diseases,Immunology,Microbiology,Parasitology
Link
https://journals.asm.org/doi/pdf/10.1128/IAI.69.1.589-592.2001
Reference21 articles.
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2. IL-4 protects against TNF-α mediated cachexia and death during acute schistosomiasis;Brunet L. R.;J. Immunol.,1997
3. Muscle wasting and dedifferentiation induced by oxidative stress in a murine model of cachexia is prevented by inhibitors of nitric oxide synthesis and antioxidants;Buck M.;EMBO J.,1996
4. An IL-13 inhibitor blocks the development of hepatic fibrosis during a T-helper type 2-dominated inflammatory response;Chiaramonte M. G.;J. Clin. Investig.,1999
5. Schistosome infection of transgenic mice defines distinct and contrasting pathogenic roles for IL-4 and IL-13: IL-13 is a profibrotic agent;Fallon P. G.;J. Immunol.,2000
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