Predominance of Th1 Immune Response in Pleural Effusion of Patients with Tuberculosis among Other Exudative Etiologies

Author:

da Cunha Lisboa Vinícius1,Ribeiro-Alves Marcelo2,da Silva Corrêa Raquel1,Ramos Lopes Isabelle1,Mafort Thiago Thomaz3,Santos Ana Paula3,Porto Amadeu Thaís1,Rufino Rogério3,Silva Rodrigues Luciana1

Affiliation:

1. Laboratory of Immunopathology, Medical Science Faculty, Rio de Janeiro State University, Rio de Janeiro, Rio de Janeiro, Brazil

2. Laboratory of Clinical Research on STD/AIDS, National Institute of Infectology Evandro Chagas, Oswaldo Cruz Institute, Rio de Janeiro, Rio de Janeiro, Brazil

3. Department of Pulmonary and Tisiology Care, Pedro Ernesto University Hospital/Universidade do Estado do Rio de Janeiro, Rio de Janeiro, Rio de Janeiro, Brazil

Abstract

Pleural tuberculosis (PlTB), a common form of extrapulmonary TB, remains a challenge in the diagnosis among many causes of pleural effusion. We recently reported that the combinatorial analysis of interferon gamma (IFN-γ), IFN-γ-inducible protein 10 (IP-10), and adenosine deaminase (ADA) from the pleural microenvironment was useful to distinguish pleural effusion caused by TB (microbiologically confirmed or not) among other etiologies.

Funder

Fundação Carlos Chagas Filho de Amparo à Pesquisa do Estado do Rio de Janeiro

Publisher

American Society for Microbiology

Subject

Microbiology (medical)

Reference45 articles.

1. World Health Organization. 2019 Global tuberculosis report 2018. World Health Organization, Geneva, Switzerland.

2. Tuberculous pleural effusion

3. Epidemiology of extrapulmonary tuberculosis in Brazil: a hierarchical model

4. Tuberculous Pleural Effusion

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