Sphingomyelin Biosynthesis Is Essential for Phagocytic Signaling during Mycobacterium tuberculosis Host Cell Entry-Reference-Cited by-同舟云学术

Sphingomyelin Biosynthesis Is Essential for Phagocytic Signaling during Mycobacterium tuberculosis Host Cell Entry

Published:2021-02-23 Issue:1 Volume:12 Page:
ISSN:2150-7511
Container-title:mBio
language:en
Short-container-title:mBio

Author:

Niekamp Patrick¹²,Guzman Gaelen¹^ORCID,Leier Hans C.¹,Rashidfarrokhi Ali¹,Richina Veronica¹,Pott Fabian¹^ORCID,Barisch Caroline³^ORCID,Holthuis Joost C. M.²^ORCID,Tafesse Fikadu G.¹^ORCID

Affiliation:

1. Department of Molecular Microbiology and Immunology, Oregon Health & Science University, Portland, Oregon, USA

2. Molecular Cell Biology Division, Department of Biology and Chemistry, University of Osnabrück, Osnabrück, Germany

3. Molecular Infection Biology Division, Department of Biology and Chemistry, University of Osnabrück, Osnabrück, Germany

Abstract

Mycobacterium tuberculosis ( Mtb ) invades alveolar macrophages through phagocytosis to establish infection and cause disease. The molecular mechanisms underlying Mtb entry are still poorly understood.

Funder

Deutsche Forschungsgemeinschaft

Division of Intramural Research, National Institute of Allergy and Infectious Diseases

Publisher

American Society for Microbiology

Subject

Virology,Microbiology

Link

https://journals.asm.org/doi/pdf/10.1128/mBio.03141-20

Reference62 articles.

1. WHO. 2018. Global tuberculosis report.

2. Mycobacterium tuberculosis: Manipulator of Protective Immunity