Contribution of Clumping Factor B to Pathogenesis of Experimental Endocarditis due to Staphylococcus aureus-Reference-Cited by-同舟云学术

Contribution of Clumping Factor B to Pathogenesis of Experimental Endocarditis due to Staphylococcus aureus

Published:2000-09 Issue:9 Volume:68 Page:5443-5446
ISSN:0019-9567
Container-title:Infection and Immunity
language:en
Short-container-title:Infect Immun

Author:

Entenza J. M.¹,Foster T. J.²,Ni Eidhin D.²,Vaudaux P.³,Francioli P.¹,Moreillon P.¹

Affiliation:

1. Division of Infectious Diseases, Department of Internal Medicine, Centre Hospitalier Universitaire Vaudois, 1011 Lausanne,1 and

2. Department of Microbiology, Moyne Institut of Preventive Medicine, University of Dublin, Trinity College, Dublin 2, Ireland2

3. Division of Infectious Diseases, Hôpital Universitaire de Genève, 1211 Geneva,3 Switzerland, and

Abstract

ABSTRACT Staphylococcus aureus Newman with an insertion mutation in clfB , the gene encoding clumping factor B, only marginally decreased infection rate ( P > 0.05) in rats with experimental endocarditis. In contrast, clfB complementation on a multicopy plasmid significantly increased infectivity ( P < 0.05) over the deleted mutants. Although clfB could affect endovascular infection, its importance in experimental endocarditis was limited.

Publisher

American Society for Microbiology

Subject

Infectious Diseases,Immunology,Microbiology,Parasitology

Link

https://journals.asm.org/doi/pdf/10.1128/IAI.68.9.5443-5446.2000

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