Helicobacter pylori Infection Induces Oxidative Stress and Programmed Cell Death in Human Gastric Epithelial Cells
Author:
Affiliation:
1. Departments of Microbiology
2. Departments of Pediatrics
3. Microbiology and Immunology, University of Texas Medical Branch, Galveston, Texas 77555
4. Medicine, University of Virginia, Charlottesville, Virginia 22908
Abstract
Publisher
American Society for Microbiology
Subject
Infectious Diseases,Immunology,Microbiology,Parasitology
Link
https://journals.asm.org/doi/pdf/10.1128/IAI.00172-07
Reference80 articles.
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2. Anasagasti, M. J., A. Alvarez, C. Avivi, and F. Vidal-Vanaclocha. 1996. Interleukin-1-mediated H2O2 production by hepatic sinusoidal endothelium in response to B16 melanoma cell adhesion. J. Cell. Physiol.167:314-323.
3. Ashktorab, H., M. Neapolitano, C. Bomma, C. Allen, A. Ahmed, A. Dubois, T. Naab, and D. T. Smoot. 2002. In vivo and in vitro activation of caspase-8 and -3 associated with Helicobacter pylori infection. Microbes Infect.4:713-722.
4. Functional Analysis of the cag Pathogenicity Island in Helicobacter pylori Isolates from Patients with Gastritis, Peptic Ulcer, and Gastric Cancer
5. Bagchi, D., G. Bhattacharya, and S. J. Stohs. 1996. Production of reactive oxygen species by gastric cells in association with Helicobacter pylori. Free Radic. Res.24:439-450.
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