Collateral Damage during Dengue Virus Infection: Making Sense of DNA by cGAS
Author:
Affiliation:
1. Department of Microbiology, Icahn School of Medicine at Mount Sinai, New York, New York, USA
2. Graduate School of Biomedical Sciences, Icahn School of Medicine at Mount Sinai, New York, New York, USA
Abstract
Funder
HHS | NIH | National Institute of Allergy and Infectious Diseases
DOD | Defense Advanced Research Projects Agency
Publisher
American Society for Microbiology
Subject
Virology,Insect Science,Immunology,Microbiology
Link
https://journals.asm.org/doi/pdf/10.1128/JVI.01081-16
Reference20 articles.
1. The emergence of arthropod-borne viral diseases: A global prospective on dengue, chikungunya and zika fevers
2. Innate Immunity Evasion by Dengue Virus
3. The dengue virus conceals double-stranded RNA in the intracellular membrane to escape from an interferon response
4. Dengue virus NS2B protein targets cGAS for degradation and prevents mitochondrial DNA sensing during infection
5. Pan-viral specificity of IFN-induced genes reveals new roles for cGAS in innate immunity
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