Chlamydia psittaci Induces Autophagy in Human Bronchial Epithelial Cells via PERK and IRE1α, but Not ATF6 Pathway-Reference-Cited by-同舟云学术

Chlamydia psittaci Induces Autophagy in Human Bronchial Epithelial Cells via PERK and IRE1α, but Not ATF6 Pathway

Published:2022-05-19 Issue:5 Volume:90 Page:
ISSN:0019-9567
Container-title:Infection and Immunity
language:en
Short-container-title:Infect Immun

Author:

Chen Li¹²,Huang Qiaoling¹³⁴,Bai Qinqin¹³⁴,Tong Ting¹³⁴,Zhou You¹³⁴,Li Zhongyu⁵,Xiao Cui¹³⁴,Chen Lili¹³⁴^ORCID

Affiliation:

1. Department of Public Health Laboratory Sciences, School of Public Health, Hengyang Medical School, University of South China, Hengyang, Hunan, China

2. Department of Laboratory Medicine, Kweichou Moutai Hospital, Zunyi, Guizhou, China

3. Hunan Key Laboratory of Typical Environmental Pollution and Health Hazards, School of Public Health, Hengyang Medical School, University of South China, Hengyang, Hunan, China

4. Hengyang Engineering Technology Research Center, Hengyang, Hunan, China

5. Institute of Pathogenic Biology, Hengyang medical school, University of South China, Hengyang, Hunan, China

Abstract

Chlamydia psittaci is an important pathogen that causes chronic and atypical pneumonia in humans. Autophagy and the unfolded protein response (UPR) are important mechanisms for regulating the growth of infectious parasitic pathogens in living cells.

Funder

Natural Science Foundation of Hunan Province

Hunan Education Commission | Scientific Research Foundation of Hunan Provincial Education Department

Fund of Hunan Key Laboratory

Publisher

American Society for Microbiology

Subject

Infectious Diseases,Immunology,Microbiology,Parasitology

Link

https://journals.asm.org/doi/pdf/10.1128/iai.00079-22

Reference48 articles.

1. Host cell death during infection with Chlamydia: a double-edged sword