Strength of Envelope Protein Interaction Modulates Cytopathicity of Measles Virus
Author:
Affiliation:
1. Molecular Medicine Program, Mayo Foundation, Rochester, Minnesota 55905
2. Immunite et Infections Virales, CNRS-UCBL UMR5537, IFR Laennec, 69372 Lyon Cedex 08, France
Abstract
Publisher
American Society for Microbiology
Subject
Virology,Insect Science,Immunology,Microbiology
Link
https://journals.asm.org/doi/pdf/10.1128/JVI.76.10.5051-5061.2002
Reference37 articles.
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2. The rule of six, a basic feature for efficient replication of Sendai virus defective interfering RNA
3. Cathomen, T., C. J. Buchholz, P. Spielhofer, and R. Cattaneo. 1995. Preferential initiation at the second AUG of the measles virus F mRNA: a role for the long untranslated region. Virology 214 : 628-632.
4. Cathomen, T., B. Mrkic, D. Spehner, R. Drillien, R. Naef, J. Pavlovic, A. Aguzzi, M. A. Billeter, and R. Cattaneo. 1998. A matrix-less measles virus is infectious and elicits extensive cell fusion: consequences for propagation in the brain. EMBO J. 17 : 3899-3908.
5. Measles Viruses with Altered Envelope Protein Cytoplasmic Tails Gain Cell Fusion Competence
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