Human B Cells Immortalized with Epstein-Barr Virus Upregulate CCR6 and CCR10 and Downregulate CXCR4 and CXCR5
Author:
Affiliation:
1. Department of Microbiology, Kinki University School of Medicine, Osaka-Sayama, Osaka 589-8511
2. Department of Tumor Virology, Institute for Genetic Medicine, Hokkaido University, Sapporo, Hokkaido 060-8638, Japan
Abstract
Publisher
American Society for Microbiology
Subject
Virology,Insect Science,Immunology,Microbiology
Link
https://journals.asm.org/doi/pdf/10.1128/JVI.76.6.3072-3077.2002
Reference19 articles.
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2. Bowman, E. P., J. J. Campbell, D. Soler, Z. Dong, N. Manlongat, D. Picarella, R. R. Hardy, and E. C. Butcher. 2000. Developmental switches in chemokine response profiles during B cell differentiation and maturation. J. Exp. Med. 191 : 1303-1318.
3. Chapman, C. J., C. I. Mockridge, M. Rowe, A. B. Rickinson, and F. K. Stevenson. 1995. Analysis of VH genes used by neoplastic B cells in endemic Burkitt's lymphoma shows somatic hypermutation and intraclonal heterogeneity. Blood 85 : 2176-2181.
4. Stable transfection of Epstein-Barr virus (EBV) nuclear antigen 2 in lymphoma cells containing the EBV P3HR1 genome induces expression of B-cell activation molecules CD21 and CD23
5. Hargreaves, D. C., P. L. Hyman, T. T. Lu, V. N. Ngo, A. Bidgol, G. Suzuki, Y. R. Zou, D. R. Littman, and J. G. Cyster. 2001. A coordinated change in chemokine responsiveness guides plasma cell movements. J. Exp. Med. 194 : 45-56.
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